Quote:
Originally Posted by Lakota
Hi everyone
Just would like some help from breeders more experienced than myself. Now I have a tendancy to over worry, so please tell me if I am being silly.
My TB mare was served in Decmeber, and had two postive preg tests (one at 18 days and one at 42 days). In March she began to produce a brown discharge. Understandably I was worried and took her to the vets, who scanned her and told me all was well and not to worry.
All has gone smoothly since then (except for the fact she has continued to produce discharge) until the other day, when the discharge increased noticably. Yesterday she began to bag up, yet she still has 10 weeks to go. Has anyone had a mare bag up this early and still go full term?
Apart from this she is in good spirits, although she is particually large for a maiden mare in foal to a pony.
Here is the mare in question. I dont have any recent pics, sorry.
Thanks for your help 
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Hi Lakota
I really hope this information below is useful.Having been involved with breeding for (a scary amount of time) I feel the info below is worth consideration.I have achieved pregnancies in mares that
every repro vet in WA has written off and live foals from mares that have exuded pus from day one post breeding.In each case, early udder development has proven to be a (either low grade or massive/hostile uterine infection).Both can cost you your pregnancy, only dirrence is early or late gestation.
Simple progesterone's or progestin's ( similar to pregnancy hormones ) DO NOT cause udder development. Other wise, why dont performance mares on regumate/CIDR/PRID/Equity have udders?
Treatment is usually simple but effective. A course of Trim-Sulfa (Only antibiotic known to cross the uterine-placental barrier and reverse uterine compromise) 1 week out of every four and P4. Please consult your EQUINE REPRO vet about this.It can make the difference.
Hope all goes well for you.
From BEFU
Mares with ascending placentitis often present in late term pregnancy with signs of premature udder development and premature
lactation. There may be a vulvar discharge. Early detection of placental problems is possible using trans-abdominal or trans-rectal
ultrasonography. Hormones such as progesterone and relaxin may be measured as indicators of foetal stress and placental failure.
Postpartum foetal membranes may be thickened and contain a fibronecrotic exudate. The region most affected is the cervical star.
Definitive diagnosis of ascending placentitis is by histopathological examination of the chorioallantoic membrane.
Ideal treatment strategies are aimed at curing the infection and prolonging the pregnancy to as close to term as possible and consist of
anti-microbials, anti-inflammatories and hormonal support.
Swabs are taken from affected mares to determine antibiotic sensitivity and to aid in treatment of foals born from these mares which
are at risk of becoming septic. If detected early enough, the chances of producing a viable foal are greatly increased.